CORRECTION OF OXIDATIVE DAMAGES DURING HEPATIC ISCHEMIA-REPERFUSION SYNDROME

Oxidative damages are a key pathogenetical mechanism of hepatic ischemia-reperfusion syndrome. The aim of the review is to analyze the current concepts of the pathogenesis of oxidative stress in the liver during ischemia-reperfusion and the most effective approaches to the correction of this pathology. The possibility of using gasotransmitters as universal modulators of the redox state in post-ischemic tissues is given particular attention.